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That was SMART goal setting because it was measurable A Be Achievable You can t lose 50 pounds in 5 days no matter what a supermarket tabloid tells you Our goals must stretch us, yes, but the stretch must be reasonable and balanced with other priorities I thought it was reasonable to collect on 95 percent of everything my sales team sold, even though they were skeptical at rst That goal motivated.

is computed using Eq. (10.13):

[49] RJ Anderson, RM Needham, A Shamir, The Steganographic File System, in Proceedings of the Second International Workshop on Information Hiding, Springer LNCS, v 1525, pp 74 84. [50] RJ Anderson, MR Roe, The GCHQ Protocol and Its Problems, in Advances in Cryptology Eurocrypt 97, Springer LNCS, v 1233, pp 134 148;

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Now we need to modify the core algorithm of SCAT to analyze networks with multiple server nodes. This modified core algorithm, which takes suitable estimates of Dirs (K) and xiv as inputs, can be described in the following steps: Cl 1 For i = 1,. . . , N and r, s = 1,. . . , R, compute estimates for the mean values Ki, (K - ls) using Eq. (9.5). Compute using Eqs. (9.7)-(9.10), for each multiple server node i, the estimated values of the marginal probabilities r;(j 1K - l,),j = 0,. . . , (mi - 2) and r = 1,. . . , R. (-1 Analyze the queueing network using one step of the MVA via Eqs. (8.38), (8.39), and (8.41). [STEP] Check the stopping condition: E;;)(K) max

150. Lewis (2000, p. 503). 151. Ibid. 152. Losick and Dworkin (1999) and Lewis (2000). 153. Chaloupka and Vinter (1996). 154. Lewis (2000, p. 504). 155. Migu lez, Hardisson, and Manzanal (1999). 156. Jones (2000). 157. Kramer (1997). He specifically calls the event of permeability transition in the membrane of the mitochondrion the central executioner, or the death switch. 158. Finkel (2001) covers the science on the status of the central role of mitochondria in programmed cell death, pros and cons. 159. Blackstone and Green (1999), Kobayashi (1998), Kroemer (1997). 160. A related issue is what are called addiction modules in some bacteria. They are certain kinds of tiny circular bits of DNA (plasmids), which lie outside the core DNA of the bacterium. The addiction modules code for a slowly decaying toxin and a fast-decaying antidote to the toxin. If the bacterium tries to oust the addiction-module, neither the toxin nor its antidote continue to be made. The antidote decays quickly. This leaves the slower-decaying toxin in the cell, killing the cell. In this way, some cells are killed, but the modules remain in those cells that allow the module to continue undisturbed. In fact, one theory is that programmed cell death in bacteria, in general, began with, and then evolved from, such addiction modules. See Yarmolinsky (1995). 161. Except in special cases. 162. Kobayashi, (1998, p. 373).

Box A2.6 (continued)

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